UNIVERSITY DAILY KANSAN
Monday, August 24,1992
7B
Teen-agers' diets are risky
The Associated Press
ATLANTA—The nation's high schools must help teen-agers cut down on greasy hamburgers and exercise more if youths are to avoid cancer and heart disease, federal health officials said Thursday.
The Centers for Disease Control, or CDC, said a survey of 77,000 high school students in 33 school systems last year found that most teen-agers are not eating properly or exercising enough.
"SCHOOLS need to offer comprehensive health education," said Laura
Kann of the CDC's Center for Disease Prevention and Health Promotion. "The exercise and nutrition behaviors that are established in youth are the types of behaviors they'll practice in adulthood."
High-fat diets and lack of exercise put adults at high risk for heart disease and cancer.
National health objectives for the year 2001 call for at least 50 percent of students to participate in daily physi- *e* & *c* activities and to reduce dietary fat consumption.
Karn said schools must push nutrition classes and offer physical educa-
courses where students spend at least 20 minutes a day exercising.
The American Cancer Society calls for 35 percent of high school students to eat five or more servings of fruits and vegetables daily and 80 percent to eat no more than two high-fat foods.
The CDC said last year's survey found only six school systems where at least half the students said they attended physical education classes every day. At least 50 percent of the students in all 33 systems did say they exercised at least 20 minutes when they were in class.
"But if that's only 20 minutes once a
week, there's a big problem," Kann said. "If you don't even go, there's no way you can be physically active."
None of the school systems met the fruit and vegetable recommendation. Schools in San Francisco, Hawaii and Pennsylvania came the closest, with 18 percent.
And only Virgin Islands students met the last recommendation, with 83 percent reporting they are low-fat foods.
The survey involved 10 big-city school systems and 23 others in 20 states, the District of Columbia, Puerto Rico and the Virgin Islands.
Study finds clues to Parkinson's
The Associated Press
LOS ANGELES—Scientists say they have taken a crucial step toward conquering Parkinson's disease by identifying a protein that is vital to healthy brain cell formation.
We are beginning to make substantial progress in an effort to understand and treat Parkinson's, said Robert Edwards, assistant professor of neurology at the UCLA School of Medicine and the study's senior author. "This could be a major key in the disease."
Parkinson's, a progressive neurological disorder that causes reduced mobility, tremors and muscular rigidity, is associated with aging but can strike young adults. There is no cure.
If the cause can be identified, Edwards said, it is possible we could prevent it and even reverse damage in people who have it.
The research also clarified an aspect of how brain cells communicate, closing a gap in scientific knowledge, Edwards said.
Mark Hallett, director of the National Institute or Neurological Disorders and Strokes in Bethesda, Md., said that the research was valuable but that it was too early to say how much could be accomplished with it.
"The more that we know about the way these cells function, the better off we are," he said.
But, based on initial reports on the research, it is not clear whether it will be relevant to Parkinson's.
According to the study, the protein — chromaffin granule amine transporter, or CGAT — helps to rid nerve cells of toxic substances that may accumulate and lead to the type of brain cell death seen in Parkinson's patients.
It could be that a lifetime of inadequate detoxification eventually leads to the disease, Edwards said.
The scientists worked with previous findings involving a toxin, MPTP, which causes symptoms similar to Parkinson's.
They took genes from adrenal glands that had proven resistant to MPTP, cloned them and transferred them to MPTP sensitive cells, and eventually identified the gene that programs for production of CGAT.
The researchers focused on brain cell communication, which occurs at junctions between cells called synapses and involves chemicals called neurotransmitters released from structures called synaptic vesicles.
The toxin responsible for Parkinson's may be dopamine, a normal brain chemical that might go awry in the disease's victims, this and other studies suggest.
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